Peroneal Neuropathy & Foot Drop Testing Sydney | Nerve Conduction Study | East Neurology

Foot Drop & Peroneal Neuropathy Testing in Sydney

Same-day nerve conduction studies to localise the lesion and predict recovery — Dr Ron Granot, FRACP, Bondi Junction

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What is Foot Drop?

Foot drop is weakness of the muscles that lift the foot upward (dorsiflexion). The toes catch on the ground with each step, producing a distinctive high-stepping ("steppage") gait. The most common cause is injury to the common peroneal nerve as it wraps around the fibular head at the side of the knee — a vulnerable, superficial location easily compressed.

Sudden onset of foot drop with severe back pain or saddle numbness may indicate cauda equina syndrome and requires emergency assessment. Otherwise, foot drop is an outpatient problem — but one where the cause matters enormously, because management of peroneal palsy at the knee is completely different from L5 radiculopathy from the back.

Common Causes

Habitual leg crossing

Prolonged pressure on the fibular head from crossed legs — particularly in people who have lost weight (reducing the protective fat pad).

Recent weight loss

Even modest weight loss reduces the cushioning around the peroneal nerve at the knee. A surprisingly common cause that often goes unrecognised.

Prolonged squat or kneel

Gardening, tile-laying, prolonged squatting (e.g. childbirth). The nerve is stretched and compressed in deep flexion of the knee.

Knee surgery or trauma

Total knee replacement, high tibial osteotomy, fibular head fracture, or post-traumatic scarring. Onset usually evident within days of the event.

Casting or splinting

Tight plaster cast or splint compressing the lateral knee. Should be released immediately if foot drop develops.

L5 radiculopathy

Compression of the L5 nerve root in the lower back can produce foot drop that mimics peroneal palsy. NCS distinguishes these.

Localising the Lesion

The pattern of weakness, sensory loss and reflex changes helps narrow down where the problem is — but only nerve conduction studies and EMG can give a definitive answer.

SiteDistinguishing clinical featuresNCS/EMG signature
Peroneal nerve at fibular headWeak dorsiflexion + eversion. Numbness over outer shin and top of foot. Spared inversion (tibialis posterior). Normal ankle jerk.Conduction block / slowing across the fibular head. EMG denervation limited to peroneal-innervated muscles.
L5 radiculopathyWeak dorsiflexion + foot inversion + hip abduction. Back pain often present. Reflexes usually preserved.NCS normal across fibular head. EMG denervation in L5-innervated muscles including tibialis posterior, gluteus medius, paraspinal muscles.
Sciatic neuropathyWeakness in both peroneal and tibial distributions (peroneal usually more affected). Sensory loss extends to sole of foot.Reduced peroneal AND tibial responses without conduction block at fibular head.
Motor neurone diseasePainless, progressive. May have fasciculations, upper motor neurone signs, or weakness in other regions.Widespread denervation on EMG involving multiple regions and roots.

See also: Sciatica & Lumbar Radiculopathy · Tarsal Tunnel Syndrome

What to Expect at Your Test

1

Brief clinical assessment

Dr Granot reviews your symptoms, the onset story (sudden vs gradual, any triggering event), and examines you — strength, sensation, reflexes and gait.

2

Nerve conduction studies

Peroneal motor recordings with stimulation at the ankle, below and above the fibular head — to detect conduction block or slowing across the knee. Tibial and sural studies to check for sciatic involvement or generalised neuropathy.

3

Needle EMG

Sampling of selected muscles to confirm and locate the lesion: tibialis anterior, extensor hallucis longus, peroneus longus, tibialis posterior (above-knee, L5-supplied), and where indicated gluteus medius and paraspinal muscles.

4

Diagnosis & prognosis

Findings discussed with you immediately. NCS at presentation predicts whether recovery is likely to be fast (intact nerve continuity) or prolonged (axonal injury). Formal report to your referring doctor the same day.

Treatment & Recovery

Most cases of peroneal palsy from compression at the fibular head recover spontaneously over weeks to months once the cause is removed. Management focuses on protecting the nerve and preventing falls during recovery.

Remove the cause

Stop leg crossing, avoid prolonged kneeling, remove or loosen tight casts. Lifestyle change is often the entire treatment.

Ankle-foot orthosis

A lightweight AFO holds the foot up during gait. Dramatically reduces falls and allows normal walking while the nerve recovers.

Physiotherapy

Stretching to prevent contracture, strengthening of supporting muscles, gait retraining. Started early, helps preserve function.

Imaging or surgery

MRI or ultrasound if a compressive lesion (ganglion, tumour) is suspected. Surgical decompression is occasionally needed for persistent compression that fails conservative measures.

Frequently Asked Questions

What causes foot drop? +
Most commonly, compression of the common peroneal nerve at the fibular head — from leg crossing, prolonged squat, weight loss, knee surgery, or casting. Other causes include L5 nerve root compression, sciatic injury, motor neurone disease, and (rarely) stroke.
How is the test useful? +
Nerve conduction studies localise the lesion — at the fibular head, in the sciatic nerve, or in the L5 nerve root. This changes management completely.
Will the nerve recover? +
Most cases of peroneal compression at the fibular head recover over weeks to months. NCS at presentation predicts recovery speed. Repeat testing at 3 months tracks reinnervation.
Should I wear a foot drop brace? +
Yes — an ankle-foot orthosis (AFO) prevents falls while the nerve recovers. It does not interfere with healing.
Do I need imaging too? +
If NCS shows the lesion at the fibular head with an obvious cause, imaging is usually not needed. If the lesion looks higher or unexplained, MRI or ultrasound may identify a ganglion or other compressive lesion.
Will I get the results on the day? +
Yes — preliminary findings discussed immediately, formal written report sent to your referring doctor the same day.

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